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Saphenous opening
Saphenous opening
Saphenous opening
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Saphenous opening
The fossa ovalis.
Front of right thigh, showing surface markings for bones, femoral artery and femoral nerve. (Fossa ovalis visible at upper right.)
Details
Identifiers
Latinhiatus saphenus, fossa ovalis femoris
TA98A04.7.03.016
TA22702
FMA58767
Anatomical terminology

In anatomy, the saphenous opening (saphenous hiatus, also fossa ovalis) is an oval opening in the upper mid part of the fascia lata of the thigh. It lies 3–4 cm below and lateral to the pubic tubercle and is about 3 cm long and 1.5 cm wide.

Structure

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Just inferolateral to the pubic tubercle the fascia extends downwards forming an arched (falciform) margin of the lateral boundary of the opening. It is covered by a thin perforated part of the superficial fascia called the fascia cribrosa which is pierced by the great saphenous vein, the 3 superficial branches of the femoral artery (except superficial circumflex iliac artery, which pierces fascia lata lateral to the saphenous opening), and lymphatics.

It transmits the great saphenous vein and other smaller vessels including the superficial epigastric artery and superficial external pudendal artery, as well as the femoral branch of the genitofemoral nerve.

The fascia cribrosa, which is pierced by the structures passing through the opening, closes the aperture and must be removed to expose it.

Clinical significance

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A femoral hernia, a protrusion of intra-abdominal organ (usually small intestine), courses through the femoral ring, femoral canal and saphenous opening sequentially.

Additional images

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  • The great saphenous vein and its tributaries at the fossa ovalis.
    The great saphenous vein and its tributaries at the fossa ovalis.
  • Superficial veins of lower limb. Superficial dissection. Anterior view.
    Superficial veins of lower limb. Superficial dissection. Anterior view.
[edit]
Revisions and contributorsEdit on WikipediaRead on Wikipedia

Saphenous opening

View on Grokipedia
from Grokipedia
The saphenous opening, also known as the saphenous hiatus or fossa ovalis, is an oval-shaped aperture in the of the that allows the passage of the from the superficial to the deep venous system. This structure measures approximately 3.75 cm in length and 2.5 cm in width, though dimensions can vary, and is covered by a sieve-like layer of called the . Positioned in the upper anterior , the saphenous opening lies just inferior to the , with its center located about 3-4 cm lateral to the . Its boundaries are formed by the superficial and deep strata of the : the superior, inferior, and lateral margins are defined by the falciform margin of the superficial stratum, while the medial boundary consists of the deep stratum. This arrangement integrates the opening into the broader fascial envelope of the lower limb, providing structural support while accommodating vascular and lymphatic transit. The primary contents of the saphenous opening include the , which pierces the to drain deoxygenated blood from the lower extremity into the , as well as efferent lymphatic vessels that connect to the superficial inguinal lymph nodes. These elements facilitate essential circulatory and lymphatic drainage, ensuring efficient return of blood and interstitial fluid from the leg to the central circulation. Clinically, the saphenous opening is significant due to its association with femoral hernias, where abdominal contents may protrude through the hiatus, leading to swelling below the and a high risk of incarceration or strangulation owing to the tight fascial boundaries. Surgical interventions in this region, such as saphenous harvesting for coronary bypass grafting, must account for its to avoid complications like vascular injury or lymphatic disruption.

Anatomy

Location and dimensions

The saphenous opening, also known as the fossa ovalis, is an oval-shaped hiatus in the covering the anterior aspect of the . It is positioned with its center approximately 3-4 cm inferior and lateral to the , placing it inferolateral to the medial end of the . This location situates the opening within the superior aspect of the , facilitating the transition of superficial structures into deeper planes of the lower limb. Typical dimensions of the saphenous opening measure about 3.75 cm in length along its long axis, which is oriented inferolaterally, and 2.5 cm in width, though these can vary between individuals. In , the opening is often marked by a subtle depression or palpable point in the region, aiding in clinical identification during procedures. Its position relates closely to key bony landmarks, including the superiorly and the superior pubic ramus via the , providing a reliable reference for anatomical orientation.

Boundaries and coverings

The saphenous opening, also known as the fossa ovalis, is bounded laterally by the falciform margin, an arched structure formed by a superior extension of the that reflects from the and adheres to the sheath of the femoral vessels. This falciform margin provides reinforcement to the opening, featuring superior and inferior cornua that enhance its structural integrity. The medial boundary is formed by the deep stratum of the , which is continuous with the surrounding of the . Superficially, the opening is covered by the , a sieve-like membranous layer of the superficial that is perforated to accommodate passing vessels and . This attaches firmly to the edges of the falciform margin, sealing the aperture while allowing selective transmission. On its deep aspect, the saphenous opening communicates directly with the and underlying , lacking additional fascial barriers beyond the continuity of the pectineal . The detailed anatomical description of the saphenous opening, including its arched falciform reinforcement and fascial layers, was first provided by Henry Gray in the 1918 edition of Gray's Anatomy, which emphasized the structural role of these boundaries in maintaining the integrity of the fascia lata.

Transmitted structures

The saphenous opening primarily transmits the great saphenous vein, which pierces the cribriform fascia to join the femoral vein at the saphenofemoral junction, allowing venous drainage from the superficial tissues of the lower limb to the deep venous system. Accompanying this vein are the superficial epigastric artery and the superficial external pudendal artery, which emerge through the opening to supply the superficial inguinal region and anterior abdominal wall. Lymphatic vessels also pass through the saphenous opening, carrying efferent drainage from the superficial tissues of the lower limb to the superficial inguinal lymph nodes. The femoral branch of the genitofemoral nerve traverses alongside these structures, piercing the fascia lata at the opening to provide sensory innervation to the skin of the upper medial thigh and external genitalia. Functionally, the saphenous opening facilitates the integration of superficial and deep venous drainage, but valvular incompetence at the saphenofemoral junction can lead to reflux and contribute to , a common cause of in the lower limb.

Development and variations

Embryological origins

The saphenous opening originates from the differentiation of al tissues that form the during early embryonic development of the lower limb, beginning around weeks 6 to 8 of . The itself derives from the embryological sheet of , particularly associated with the developing , which provides the foundational layer for the thigh's deep investing . This mesodermal origin establishes the structural framework through which the opening later forms, as the thickens and organizes around the emerging musculature of the . The formation of the saphenous opening results from the differential growth and migration of the great saphenous vein primordium through the developing thigh fascia, allowing the superficial vein to pierce and connect with the deep venous system. The great saphenous vein emerges around weeks 7 to 8 from the pre-axial venous plexus, guided by the femoral nerve during the third stage of lower limb venous organogenesis. Concurrently, the lower limb bud undergoes medial rotation between weeks 6 and 8, repositioning the superficial venous network—including the great saphenous vein primordium—to its adult medial orientation and facilitating the establishment of connections to the deep veins at the future sapheno-femoral junction. By the 12th week, the vein matures further, integrating with the femoral vein proximal to the opening. The cribriform fascia, which covers the saphenous opening, develops from thickened connective tissue that fills the fossa ovalis region, as demonstrated in embryological studies of human fetuses from 11 to 35 weeks of gestation; this lamina cribrosa reinforces the hiatus while permitting passage of the great saphenous vein and associated structures. No specific genetic markers have been identified for the saphenous opening in the literature, with its development instead linked to broader processes of fascial differentiation and vascular patterning in the lower limb mesoderm. Embryological descriptions of the saphenous opening remained limited until the 20th century, with foundational insights from comparative anatomy and early human fetal studies.

Anatomical variations

The saphenous opening, also known as the fossa ovalis, demonstrates notable anatomical variations in position, size, and associated venous structures at the saphenofemoral junction, observed in cadaveric dissections, surgical explorations, and imaging studies such as duplex . These deviations influence venous drainage patterns and surgical planning. Positional variations are common, with the opening typically located 2.5-3.5 cm inferolateral to the . In a comparative study of 50 cadaveric limbs and 100 surgical patients with , the mean distance from the to the saphenofemoral junction was 3.1 ± 0.5 cm in cadavers and 2.8 ± 0.8 cm in patients, with a significant positive correlation to length (r=0.58-0.6, p<0.001). Similarly, measurements in 150 South Indian adults yielded a mean distance of 3.04 ± 0.4 cm from the , varying by age group from 2.52 cm in those under 20 years to 3.19 cm in those aged 41-50 years, without significant sex or side differences. Size anomalies include variations in the oval-shaped opening's dimensions, normally measuring about 3.75 cm in length and 2.5 cm in width, or exhibit irregular shapes. Cadaveric studies report great variability in size and shape, with the center positioned at a mean 3.53 ± 0.38 cm from the pubic tubercle. Venous variations at the saphenofemoral junction frequently involve the great saphenous vein (GSV), with duplication occurring in 9.6% of cases as a bifid junction and 1.7% as two separate junctions, based on a meta-analysis of 16 studies encompassing 7,433 limbs. Atypical drainage patterns, such as the GSV crossing posterior to the common femoral artery (0.12%) or passing between the profunda and superficial femoral arteries (0.08%), are less common but documented in ultrasound assessments of 2,552 limbs. In cadaveric dissections of 75 limbs, GSV duplication was noted in 2.7%, with tributaries numbering 0-7 (mean 3.8), most often 4-5 (61.3%), and 20% draining directly into the common femoral vein rather than the junction. These configurations arise from incomplete fusion during embryological development of the venous system. Such variations can contribute to venous insufficiency by disrupting normal flow dynamics.

Clinical significance

Relation to femoral hernias

The saphenous opening, also known as the fossa ovalis, serves as the distal exit point for femoral hernias, which develop when intra-abdominal contents protrude through the femoral canal—a narrow passage medial to the femoral vein bounded by the inguinal ligament superiorly, the lacunar ligament medially, and Cooper's ligament inferiorly. In this pathology, omentum or loops of small intestine typically herniate inferiorly through the femoral ring, descend along the femoral canal, and emerge subcutaneously via the saphenous opening, where they become covered only by the cribriform fascia and skin. This emergence often results in a visible or palpable bulge in the upper thigh or groin, as the hernia sac expands beyond the confines of the canal. The condition arises from congenital weakness or acquired factors such as increased intra-abdominal pressure, obesity, or pregnancy, leading to enlargement of the femoral ring. Femoral hernias account for approximately 3% of all hernias, though this proportion rises significantly in s, comprising up to 20-30% of hernias in women due to the wider , which enlarges the femoral canal relative to the . They are 4-10 times more common in females than males and increase in incidence with age, particularly after 50 years. Symptoms often include a small, tender swelling in the inferolateral to the , which may worsen with coughing, straining, or standing and reduce when lying down; however, up to one-third of cases are until complications arise. Diagnosis is primarily clinical, with the bulge distinguished by its position below the , but or CT imaging is recommended for confirmation, especially in obese patients or to assess for incarceration or strangulation, which occurs in 15-20% of cases due to the narrow femoral canal compressing the hernia contents. Femoral hernias were first systematically recognized in the , with early anatomical descriptions by figures like Lorenz Heister and Franz Hesselbach, who detailed their passage through the . Surgical repairs evolved from simple high ligation of the sac in the to more robust techniques in the , including McVay's Cooper repair () and the introduction of reinforcement in the 1950s by Francis Usher using to buttress the femoral opening and reduce recurrence rates to under 5%. Modern approaches often employ laparoscopic or open preperitoneal placement to reinforce the and surrounding structures. Unlike inguinal hernias, which pass through or above the supero-medial to the , femoral hernias traverse deep to the via the femoral canal and saphenous hiatus, positioning the bulge infero-lateral to the and conferring a higher of strangulation due to the rigid boundaries. This anatomical distinction is critical for preoperative differentiation, as misdiagnosis can lead to inappropriate surgical incisions.

Role in venous surgery

The saphenous opening plays a central role in surgical interventions for (GSV) incompetence, a common cause of and , as it marks the site where the GSV joins the at the saphenofemoral junction. These procedures are particularly relevant for the approximately 25% of adults affected by , where GSV reflux contributes significantly to symptoms like pain, swelling, and skin changes. Saphenofemoral ligation remains a standard open surgical approach for treating GSV incompetence, involving a incision directly over the saphenous opening to access and tie off the junction, thereby preventing retrograde flow while preserving deep venous drainage. This technique effectively addresses transmitted venous structures through the opening, such as the GSV, and is often combined with vein stripping for complete elimination. Endovenous techniques have largely supplanted traditional for accessing and treating the GSV via the saphenous opening, including thermal (radiofrequency or ) and , which seal the vein from within under guidance to minimize risks like injury. In these procedures, a is advanced through the GSV to the saphenofemoral junction at the opening, where energy or sclerosant is applied to induce endothelial damage and vein closure. Complications associated with interventions at the saphenous opening include formation due to disruption of nearby lymphatic vessels and wound infections from the incision site, occurring in up to 3-5% of cases. Anatomical variations, such as duplicate GSVs traversing the opening, can heighten procedural complexity by necessitating additional imaging and ligation sites, potentially increasing operative time and recurrence risk. As of 2025, guidelines from the Society for Cardiovascular Angiography and Interventions conditionally recommend endovenous ablation therapy, including , for symptomatic reflux at the saphenous opening, which is associated with high long-term closure rates (typically above 90%) and faster recovery (often within days) compared to open ligation (1-2 weeks or more).

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